Cure and prevent diabetes mellitus with diet, not drugs
Part 7: Type 1 Diabetes
Type-1 diabetes normally affects young people, commonly around the ages of ten
or twelve, although it can occur as early as one year and as late as forty. The
disease tends to develop rapidly and is severe. In this form of the disease,
the beta cells of the pancreas do not produce enough insulin. This type of
diabetes is called either type-1 diabetes or, more technically, insulin
dependent diabetes mellitus (IDDM).
Two kinds of problems occur when the body doesn't make sufficient insulin:
- Hyperglycemia occurs when blood glucose levels get too high. This can occur when the body gets too little insulin or there is too much glucose in the bloodstream. Untreated, hyperglycemia may develop into ketoacidosis, a very serious condition. Treatment is invariably with insulin injections to make up the shortfall and reduce blood glucose levels.
- Hypoglycemia is the exact opposite of hyperglycemia. This occurs when blood glucose levels get too low, when the body gets too much insulin or too little food. Hypoglycemia is the most common problem in children with diabetes usually caused by diabetic insulin overdose, a missed meal or unexpected exercise. Usually it is mild and is easily treated by giving the child something sweet. It is hypoglycaemia, however, that is dangerous if left untreated as it can lead to coma and death.
EARLY SYMPTOMS OF HYPOGLYCAEMIA:
- Feeling shaky or irritable
- Feeling dizzy or lightheaded
- Feeling hot, followed by excessive sweating
- Blurred vision and/or slurred speech
- Tingling or numbness in the mouth or lips
- Headache
(Hypoglycaemia also affects people who are not type-1 diabetics. An example of the effects of this is given here )
WHAT IS HYPOGLYCAEMIA?
Hypoglycemia occurs when the blood sugar levels are abnormally low. In some
cases, hypoglycemia can cause a person to become aggressive or seem
uncooperative, which can easily be mistaken for drunkenness by people who do
not know about the effects of hypoglycemia. In extreme cases, hypoglycemia can
cause a person to become unconscious. If this happens to someone you are with,
seek medical assistance immediately and inform those providing treatment that
the person has diabetes.
Type-1 can be induced by anything that causes the beta cells in the pancreas to
malfunction. This could be a physical trauma, infectious disease, allergy,
autoimmune disease or tumour. Generally, however, type-1 is believed to be an
inherited form of the disease as it is more likely to occur in people who have
close relatives with diabetes. But this seems unlikely, as type-1 diabetes is
not found in the animal kingdom either in meat or plant eating animals, where
those animals live in their natural habitat. Neither does type-1 diabetes exist
amongst peoples who have not had extensive contact with the industrialised
societies: the Inuit, Maasai, Hunza, and other indigenous peoples whose diets
are typically low in carbohydrates.
1
While not a single case of type-1 diabetes has been found among the meat- and
fat-eating Inuit population of Alaska, there have been cases of the maturity
onset type of diabetes.
2
These appear to be the result of increasing carbohydrates introduced into the
modern Inuit diet by 'civilisation'.
As diabetes is wholly restricted to peoples of Western industrialised
civilisation, it cannot have a genetic origin, except insofar as peoples with
differing evolutionary backgrounds do have differing levels of the disease.
Maternal diet
Family dietary traits and lifestyle can play a major part in the appearance of
type-1 diabetes within families.
If a pregnant woman eats too much carbohydrate, this will raise her insulin
levels. It is not thought that insulin itself crosses the placenta from mother
to foetus. However, insulin produces antibodies that do.
3
Once in the foetus these increase glycogen and fat deposits resulting in an
abnormally large baby. It may predispose that baby to type-1 diabetes.
Birth weight is also predictive of future diabetes. A Norwegian population
based cohort study by record linkage of the medical birth registry and the
National Childhood Diabetes Registry looked at all live births in Norway
between 1974 and 1998 (1,382,602 individuals).
4
Over a maximum of 15 years of observation, a total of 8 184 994 person years
of observation in the period 1989 to 1998, 1824 children with type 1 diabetes
were diagnosed between 1989 and 1998. There was a direct linear increased
incidence of type 1 diabetes with increasing birth weight. It was relatively
weak but significant. The rate ratio for children with birth weights 4500 g or
more was 2.21 times as many as compared with those with birth weights less than
2000 g.
Thus, the way an expectant mother eats can be expected to have an effect on the
future health of her offspring. She — I say 'she' because mother usually
controls a family's food — will also influence the way her children eat.
They usually eat the way she does so it is important that mother sets a good
example.
Conventional treatment
The medical profession generally regards type-1 diabetes is incurable. It is
managed conventionally with a carbohydrate-based, low-fat diet. As the
carbohydrates in such a diet inevitably put large amounts of glucose in the
bloodstream, daily insulin injections have to be administered to bring these
high levels of glucose in the blood down to normal. For the patient, this means
walking a tightrope for life, as exactly the right amount of insulin must be
given or it will either reduce glucose levels too much or not enough. As we saw
earlier, insulin supplementation is a serious health hazard.
But the Type-1 diabetic rarely produces no insulin at all. Even in severe
cases, at the time of initial diagnosis five to fifteen percent of the
pancreas's beta cells usually survive to produce insulin. If these are relieved
of the burden of continually having to reduce excessive levels of blood
glucose, they can usually produce sufficient insulin for the variety of other
metabolic processes that need it.
There is a better way
A Polish doctor, Jan Kwasniewski, has successfully treated type-1 diabetics for
over thirty years merely by reducing their carbohydrate intake to 'an amount
dictated by the insulin-producing capacity of the sufferer'.
5
This amount, he says, typically equates to 1.5 grams of carbohydrate per
kilogram body weight for a growing child and between forty and fifty grams for
an adult. With this regime, the main energy source is dietary animal fat. On
such a diet, his type-1 diabetic patients no longer need to use insulin.
But is is essential that this dietary treatment is started immedately
as, if it is not begun as soon as diagnosis is confirmed, the beta cells will
continue to deteriorate and, once they are lost, they never recover.
The dietary regime is similar to that in
Part 6
of this series. The basic principle is to reduce carb intake (and so reduce
insulin requirement) and allow the body to burn fats as its primary energy
source. But be aware that proteins as well as carbs can raise blood glucose
levels. For this reason, the cut-back on carbs
must
be made up with fats — NOT proteins.
The type-1 diabetic is in quite a different position from the type-2. By
definition, there will be little beta cell activity and all type-1 diabetics
differ in their insulin output. Thus this dietary regime, just like any other,
must be monitored carefully, at least at first until its effects are known. If
there is some insulin being produced it may be possible to stop injecting
altogether. If there is none, you will still have to inject — but you
will inject less.
References
1
. Yudkin J. Evolutionary and historical changes in dietary carbohydrates.
Am J Clin Nutr.
1967; 20: 108-115.
2
.
JAMA
March 27, 1967
3
. Menon R K, et al. Transplacental passage of insulin in pregnant women with
insulin dependent diabetes mellitus: its role in fetal macrosomia.
N Eng J Med
1990; 323: 309-15
4
. Stene LC, Magnus P, Lie RT, et al. The Norwegian Childhood Diabetes Study
Group. Birth weight and childhood onset type 1 diabetes: population based
cohort study.
BMJ
2001; 322 : 889-892
5
. Kwasniewski J, Chylinski M.
Homo Optimus.
Wydawnictwo WGP, Warsaw, 2000: 163-6.
Introduction
Part 1: The scale of the problem
Part 2: What is diabetes -- Are you at risk?
Part 3: Conventional treatment for Type-2 diabetes – and why it fails
Part 4: Why carbs are the wrong foods for diabetics
Part 5: The evidence
Part 6: The correct diet for a Type-2 diabetic, (or treatment without drugs)
Part 7: Treatment for Type-1 diabetes
Suitable foods for diabetics
Last updated 6 February 2008
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